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When the walls of these arteries are distended by an increase in blood pressure, the baroreceptors are stimulated, and information is transmitted to the brain via the parasympathetic nervous system.The parasympathetic system then responds (via the vagus nerve) by increasing the release of acetylcholine at the SA node, thereby decreasing heart rate and cardiac output.The nervous system is one of the major influences on the heart that allows it to respond rapidly to the changing requirements of the body’s tissues.

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Stimulation of the vagus nerves releases the neurotransmitter acetylcholine, which increases the permeability of cardiac cell membranes to potassium.

This permits leakage of K ions to the outside, creating a state of hyperpolarity in which the cells are less easily excited.

Norepinephrine increases the permeability of myocardial cells, including pacemaker cells and contractile tissue, to sodium and calcium.

The increase in sodium permeability lowers the threshold potential of SA node cells, causing them to fire more rapidly.

At any given time, the effect of the nervous system on the heart is the net balance between the opposing actions of the sympathetic and parasympathetic systems.

Cardiac Acceleration- the Sympathetic Pathway In response to stress or exercise, the sympathetic nerves release the hormone norepinephrine.

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